Copper is an essential element that is a cofactor of many enzymes. Copper metabolism is disturbed in Wilson's disease, Menkes diseases, primary biliary cirrhosis, and Indian childhood cirrhosis. Copper concentrations increase in acute phase reactions. Copper concentrations are decreased with nephrosis, malabsorption, and malnutrition. Copper concentrations are also useful to monitor patients, especially preterm newborns, on nutritional supplementation. Results of copper are often interpreted together with ceruloplasmin.
* This volume does not allow for repeat testing
Centrifuge and separate plasma from cells within 2 hours of collection
Transfer plasma to an acid washed or metal-free plastic vial
Hemolysis
Not separated from cells
Submitted in non-trace metal container
Submitted in non acid washed container
Transport
< 6 months: 38 - 104 mcg/dL
< 12 months: 24 - 152 mcg/dL
< 2 years: 76 - 193 mcg/dL
< 4 years: 87 - 187 mcg/dL
< 6 years: 56 - 191 mcg/dL
< 10 years: 117 - 181 mcg/dL
< 14 years: 87 - 182 mcg/dL
< 18 years: 75 - 187 mcg/dL
>= 18 years: 70 - 175 mcg/dL
Copper is an essential element that is a cofactor of many enzymes. Copper metabolism is disturbed in Wilson's disease, Menkes diseases, primary biliary cirrhosis, and Indian childhood cirrhosis. Copper concentrations increase in acute phase reactions. Copper concentrations are decreased with nephrosis, malabsorption, and malnutrition. Copper concentrations are also useful to monitor patients, especially preterm newborns, on nutritional supplementation. Results of copper are often interpreted together with ceruloplasmin.
1 to 2 days
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