Copper is an essential element that is a cofactor of many enzymes. Copper metabolism is disturbed in Wilson's disease, Menkes diseases, primary biliary cirrhosis, and Indian childhood cirrhosis. Copper concentrations increase in acute phase reactions. Copper concentrations are decreased with nephrosis, malabsorption, and malnutrition. Copper concentrations are also useful to monitor patients, especially preterm newborns, on nutritional supplementation. Results of copper are often interpreted together with ceruloplasmin.
2.0 mL Serum from a Navy Blue Top Tube - No Additive, trace element serum red striped label, in an Serum Trace Element and Metal Free Plastic Tube
2.0 mL Plasma from a Navy Blue Top Tube - EDTA, in an Metal Free
5 days Ambient
10 days Refrigerated
30 days Frozen
Centrifuge and separate from cells within 2 hours of collection
Transfer separated serum/plasma to a plastic acid washed or metal free vial
0.7 mL Serum
< 6 months: 38 - 104 mcg/dL
< 12 months: 24 - 152 mcg/dL
< 2 years: 76 - 193 mcg/dL
< 4 years: 87 - 187 mcg/dL
< 6 years: 56 - 191 mcg/dL
< 10 years: 117 - 181 mcg/dL
< 14 years: 87 - 182 mcg/dL
< 18 years: 75 - 187 mcg/dL
>= 18 years: 70 - 175 mcg/dL
Copper is an essential element that is a cofactor of many enzymes. Copper metabolism is disturbed in Wilson's disease, Menkes diseases, primary biliary cirrhosis, and Indian childhood cirrhosis. Copper concentrations increase in acute phase reactions. Copper concentrations are decreased with nephrosis, malabsorption, and malnutrition. Copper concentrations are also useful to monitor patients, especially preterm newborns, on nutritional supplementation. Results of copper are often interpreted together with ceruloplasmin.
4 to 7 days
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